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dimanche 30 juin 2013

Is industry able to end obesity?

Repas paléo

Chez mon ami Pierre un petit repas vite fait avec des aliments pas des produits !
Avec des transformations minimales pas des destructions massives au feu ou bien aux ondes donc un maximum de micro nutriments!
Le petit lait de chèvre fermenté et pétillant est une contribution a la flore et une source d'acides aminés branches sans lactose!

lundi 17 juin 2013

Nutritional emergency: stop high GI foods and foods products!

DRAFT Scientific Consensus Statement*

  1. Carbohydrates present in different foods have distinct physiological effects, including effects on post-prandial glycemia (PPG), with different implications for health.
  1. Reducing PPG is recognized as a beneficial physiological effect.
  1. Ways to reduce PPG include slowing carbohydrate absorption by consuming low glycemic index (GI) and glycemic load (GL) foods to reduce the dietary GI and GL.
  1. The GI methodology is a sufficiently valid and reproducible method for differentiating foods based on their glycemic response (GR) [footnote: high vs low GI foods as defined by the isostandard, [55; processing and cooking effects]
  1. The GI quantifies specific physiological properties of carbohydrate -containing foods as influenced by the food matrix. These characteristics extend beyond their chemical composition including delaying gastric emptying and reducing the rate of digestion and small intestinal absorption.
  1. When considering the macronutrient composition, the GL (the product of GI and carbohydrate content/1000kJ) is the single best predictor of the glycemic response of foods.
  1. There is convincing evidence from meta-analyses of controlled dietary trials that diets low in GI improve glycemic control in people with type 2 diabetes.
  1. There is convincing evidence from meta-analyses of prospective cohort studies that low GI/GL diets reduce the risk of type 2 diabetes.
  1. There is convincing evidence from a large body of prospective cohort studies that low GI/GL diets reduce the risk of coronary heart disease.
  1. The proof of principle for the concept of slowing carbohydrate absorption is the use of alpha-glucosidase inhibitors (acarbose etc.) to reduce progression to type 2 diabetes and coronary heart disease.
  1. The carbohydrate quality as defined by GI/GL is particularly important for individuals who are sedentary, overweight and at increased risk of type 2 diabetes.
  1. Potential mechanisms for reduction of type 2 diabetes include evidence that low GI/GL diets improve insulin sensitivity and beta-cell function in people with type 2 diabetes and those at risk for type 2 diabetes.
  1. Potential mechanisms for reduction of coronary heart disease include evidence that low GI/GL diets improve blood lipids and inflammatory markers including C-reactive protein (CRP).
  1. Probable evidence exists for low GI/GL diets in body weight management.
  1. The GI complements other ways of characterizing carbohydrate-foods, such as fiber and whole grain content.
  1. Low GI is to be considered in a context of a healthy diet.
  1. Given the rapid rise in diabetes and obesity there is a need to communicate information on GI/GL to the general public and health professionals.
  1. This should be supported by inclusion of GI/GL in dietary guidelines and in food composition tables.
  1. In addition package labels and low GI/GL symbols on healthy foods should be considered.
  1. More comprehensive high-quality food composition tables need to be developed for GI/GL at the national level.
*NOTE:  This Statement will be finalized when footnotes and scientific references and other minor changes are added.

Scientific Consensus Committee:

David J.A. Jenkins, MD, PhD, DSc, University Professor and Canada Research Chair in Nutrition  and Metabolism,  Department of Nutritional Sciences,  Faculty of Medicine, University of Toronto, Director, Risk Factor Modification Centre, St. Michael’s Hospital (Toronto, Canada)
Walter C. Willett, MD, DrPH, Fredrick John Stare Professor of Epidemiology and Nutrition Chair, Department of Nutrition, Harvard School of Public Health (Boston, USA)

Livia Augustin, PhD,  Research Fellow, Risk Factor Modification Centre, St. Michael’s Hospital (Toronto, Canada)
Sara Baer-Sinnott, President, Oldways (Boston, USA)
Alan W. Barclay, PhD, Head of Research, Australian Diabetes Council; Chief Scientific Officer Glycemic Index Foundation (Sydney, Australia),
Inger Björck, PhD, Professor and Managing Director Antidiabetic Food Centre, Lund University (Lund, Sweden)
Jennie C. Brand-Miller, PhD, Professor, Boden Institute of Obesity, Nutrition, Exercise and Eating Disorders, University of Sydney (Sydney, Australia)
Furio Brighenti, DrPH, Professor of Human Nutrition, Department of Food Science University of Parma (Parma, Italy)
Anette E. Buyken, PhD, Research Associate, Department of Nutritional Epidemiology, University of Bonn (Bonn, Germany).
Antonio Ceriello, MD, Head of Research at the Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS) (Barcelona, Spain)
Cyril W.C. Kendall, PhD, Research Associate, Department of Nutritional Sciences, Faculty of Medicine, University of Toronto (Toronto, Canada) and Adj. Professor College of Pharmacy and Nutrition, University of Saskatchewan (Saskatoon, Canada)
Carlo La Vecchia, MD, Chief, Department of Epidemiology, Mario Negri Institute, and Professor of Epidemiology, University of Milan, (Milan, Italy)
Geoff Livesey, PhD, Director, Independent Nutrition Logic (Wymondham, UK)
Simin Liu, MD, ScD, Professor, Departments of Epidemiology and Medicine, Brown University (Providence, USA)
Andrea Poli, MD, Scientific Director, Nutrition Foundation of Italy (Milan, Italy)
Gabriele Riccardi, MD, Full Professor of Endocrinology and Metabolic Diseases, Department of Clinical Medicine and Surgery, Federico II University (Naples, Italy)
Salwa W. Rizkalla, MD, PhD, DSc, Senior Researcher, National Institute of Health and Medical Research (INSERM) U 872, team 7, Research centre in human nutrition, ICAN Institute of Cardiometabolism & Nutrition, University Pierre et Marie Curie-Paris 6, Centre of Research in Human Nutrition, Pitié Salpêtrière Hospital (Paris, France).
John L. Sievenpiper, MD, PhD, Toronto 3D Knowledge Synthesis and Clinical Trials Unit, Clinical Nutrition and Risk Factor Modification Centre, St. Michael's Hospital (Toronto, Canada), Department of Pathology and Molecular Medicine, Faculty of Health Sciences, McMaster University (Hamilton, Canada).
Antonia Trichopoulou, MD, PhD, Professor and Director, World Health Organization  Collaborating Centre for Food & Nutrition,  Department of Hygiene and Epidemiology, University of Athens Medical School, and Vice President, Hellenic Health Foundation (Athens, Greece)
Thomas M.S. Wolever MD, PhD, Professor, Department of Nutritional Sciences, University of Toronto (Toronto, Canada)

Early diet is of major importance for future health of your kids

Early Diet Impacts Infant Rhesus Gut Microbiome, Immunity, and Metabolism

Department of Nutrition, §Department of Food Science and Technology, One Shields Avenue,University of California, Davis, Davis, California 95616, United States
 Fonterra Ingredients Innovation, Fonterra Co-operative Group, Private Bag 11029, Fitzherbert Dairy Farm Road, Palmerston North, New Zealand
J. Proteome Res.201312 (6), pp 2833–2845
DOI: 10.1021/pr4001702
Publication Date (Web): May 7, 2013
Copyright © 2013 American Chemical Society
*E-mail: Ph: (530) 752-6804. Fax:(530) 752-8966.

"Epidemiological research has indicated a relationship between infant formula feeding and increased risk of chronic diseases later in life including obesity, type-2 diabetes, and cardiovascular disease. The present study used an infant rhesus monkey model to compare the comprehensive metabolic implications of formula- and breast-feeding practices using NMR spectroscopy to characterize metabolite fingerprints from urine and serum, in combination with anthropometric measurements, fecal microbial profiling, and cytokine measurements. Here we show that formula-fed infants are larger than their breast-fed counterparts and have a different gut microbiome that includes higher levels of bacteria from the Ruminococcus genus and lower levels of bacteria from the Lactobacillus genus. In addition, formula-fed infants have higher serum insulin coupled with higher amino acid levels, while amino acid degradation products were higher in breast-fed infants. Increases in serum and urine galactose and urine galactitol were observed in the second month of life in formula-fed infants, along with higher levels of TNFα, IFN-γ, IL-1β, IL-4, and other cytokines and growth factors at week 4. These results demonstrate that metabolic and gut microbiome development of formula-fed infants is different from breast-fed infants and that the choice of infant feeding may hold future health consequences."

jeudi 13 juin 2013

If you eat sugars you have to exercise, a study shows.

Three 15-min Bouts of Moderate Postmeal Walking Significantly Improves 24-h Glycemic Control in Older People at Risk for Impaired Glucose Tolerance

  1. William Rumpler, PHD2
+Author Affiliations
  1. 1Department of Exercise Science, The George Washington University School of Public Health and Health Services, Washington, DC
  2. 2Beltsville Human Nutrition Research Laboratory, United States Department of Agriculture, Beltsville, Maryland
  1. Corresponding author: Loretta DiPietro,

We should not forget that all people are at risk for impaired glucose tolerance.

mercredi 12 juin 2013

Palm oil: the global commercial war about fats

For several months a number of articles dealing with the subject came from the use of palm oil in food products in industrialized countries. This debate was first initiated by environmentalists on the grounds that this production would be a danger to the ecological systems of the producing countries. Like all those environmental issues of palm oil does not escape the simplification or even simplistic ideological approaches. This is actually a very complex agricultural science subject, ecological and industrial as may be encountered in the climate debate, shale gas or GMOs ...
There can be no truth in this area but data, interpretations of changes based on the results observed in short a complex and dynamic system that producers, regulators, markets are changing from year to year. The theory of profit fall guilty of oversimplification, palm oil and its producers are certainly not guilty designated our environmental situation.


Families to cook, industrial for their food products need fat.
Palm oil is a vegetable fat. Production worldwide has increased by about three from 2000 to 2010. Europe and within many countries including France are big dairy and oilseeds (sunflower, olive, soybean, canola, flax). This fat production competes with other animal or vegetable fats global market with oil palm. France was the largest producer of oilseeds in the EU in 2007(
Italy followed Greece and Spain was the largest producer of olive oil in 2007. Regarding dairy fats in France in 2007 was still the second EU country for the number of dairy cows. Palm oil (ME 556) is, in value, the second largest import item fats in the EU in 2010 after soybean meal (ME 6255). In this area after each defends his bold and it is natural for all should not forget these issues in the debate.
(Http:// oil is a production that is accused of having caused the deforestation of primary forests in countries where few other resources exist. This is the exact time and arrogant. How industrialized countries also have deforested centuries ago their respective countries could have a self-proclaimed legitimacy to limit the cultivation of oil palm in Indonesian or Malay? But there are also industrial issues. Consumption of palm oil by the food industry is currently difficult to replace because of lower yields on other oilseeds (it takes more agricultural land for the same amount of fat with sunflower or soybean). This is why the process of environmental palm oil seems to us unfounded and dependents.

Therefore the idea of a tax is obviously very suspicious of protectionism.
Remember that there is no nutritional tax, but only food taxes. They are paid by the consumer that the charge is paid to the production or consumption. Food taxes are experienced around the world for many yearse2931).. it has been amply demonstrated that food taxes have very little effect on human behavior (BMJ 2012, 344:  Provided it could be a perfect economic justification for a food tax: offset the cost of negative externalities paid by the community as a result of agricultural, industrial or degradation of the health of consumers of the food in question on fatty foods affect health (read weight decrease) occurs only from a tax of 20% of the market price. In the literature the most effective food taxes are those on sugary drinks. This is why a tax on palm oil appropriately imposed on the food industry but paid by consumers without being able from the past experiences in hope for a health benefit is simply a trade barrier not paid by importers but by the French.


Accusing saturated fats in industrialized countries is unwise on scientific available data
It is clear that we have no consensus on the effects of palm oil on the health of people who consume. The many studies are contradictory. The paradigm that saturated fats are the cause of cardiovascular disease is largely dented. Palm oil is a topic of growing interest in the medical community since 2000 publications are 40-50 articles per year but in 2011 it increased to 129, then 154 and in 2013, 73 articles have been published . What is well established is that the first intake of saturated fat is not the cause of diseases related to atherosclerosis. The atheroma is a multifactorial polygenic disease and the risk is predicted with much greater reliability by an equation with several variables rather than the single digit of LDL cholesterol (bad cholesterol improperly named). In this equation smoked tobacco, heredity, high blood pressure, physical inactivity and obesity or diabetes are incomparably more important in augmenation risk. The second point is related to factual refining fats. In the same way as olive oil unfiltered richer in phenols and vitamin E contributes significantly to the benefits of the Mediterranean diet, the red palm oil (unrefined) is extremely rich in fats and vitamin E in antioxidants which is very favorable to health especially in developed countries where industrial food is poor in micronutrients. The third point is firmly supported by numerous and consistent work is the harmful effects of trans fatty acids from thermal hydrogenation of vegetable fats whatsoever. Again this is not the fat that is in question but its transformation which raises highly atherogenic fatty acids. Say that consume palm oil also contains 30% oleic acid (olive oil) is a public health problem is not scientifically established.

Consider the example of Nutella
In two tablespoons of Nutella there are 37 g of product. 4g saturated fat or 36 calories. For individuals at risk of total calories from saturated fat in the day should not exceed 140 calories, we are far from this recommandation (
More importantly there are 21 grams of sugar is 84 calories and is refined sucrose (white table sugar beet) added a sugar high glycemic index of 80. The sugar is sucrose consisting of glucose and fructose. Glucose can be used by our cells after insulin was secreted by the pancreas in response. It circulates in the blood. The fructose is absorbed up by the liver and contributes to elevate triglycerides and LDL cholesterol particularly in sedentary individuals. Nutella is therefore 57% of refined beet sugar, also an inexpensive quick sugar.
It is more rational to ask metabolic consequences of this contribution of fast sugar (insulin resistance, obesity, diabetes) than that of 4g saturated fats especially in the perspective of obesity, metabolic syndrome and Type 2 diabetes.
Finally it is always rational to focus on the fact that palm oil is refinedused depriving consumers of Nutella Vitamin A and E and phenols naturally present in red palm oil . Nutella is singularly deficient in micronutrients.

This tax and purpose seem particularly unclear on the facts.
This tax is potentially a source of revenue for the state. It is very unlikely in the light of scientific evidence that it improves the environment or the health of people who consume. Why is this will of the executive? It is political opportunism and the most important daily lobbies and the press. It is regrettable that the environmental aspects are reported in the press that a unilateral point of view, the environmental organizations in developed countries. A more balanced and fair perspective would improve the quality of fats in our diet incorporated rather than scapegoats. About the prevention of cardiovascular disease we can not escape questioning past about saturated fat truths, black magic of “bad cholesterol” and the effectiveness of changes in habits of life compared the drug approaches. Eventually, the food industry should instead wait for a regulation banning anticipate stopping to add trans fats to their products and improve the quality of fats using unrefined fats.

Epigenetics of human obesity